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There are parallels between visual attention and eye movements because both serve the purpose of directing our processing of visual information to stimuli from a region in space that is small enough to handle for our brain.

Since visual attention and eye movements are so tightly connected in the process of visual exploration of a scene, it has been suggested that the same mechanisms may be (partially) responsible for guiding them.

There is evidence suggesting that one cannot plan a saccade to one point in space and turn covert visual attention to another at the same time.

Born et al. provided evidence which shows that preparing a saccade alone already enhances visual processing at the target of the saccade: discrimination targets presented before saccade onset were identified more successfully if they were in the location of the saccade target than when they were not.

Born et al. showed that, if the color of a saccade target stimulus is task relevant, then identification of a discrimination target with that same color is enhanced even if it is not in the same location.

Saccades evoked by electric stimulation of the deep SC can be deviated towards the target of visual spatial attention. This is the case even if the task forbids a saccade towards the target of visual spatial attention.

FEF stimulation elicits saccadic eye movements.

Presaccadic activity is not measured in FEF for spontaneous saccades but for purposive saccades.

The motor map is not monotonic across the entire FEF, but sites that are close to each other have similar characteristic saccades.

Certain neurons in the deep SC emit bursts of activity before making a saccade.

It has long been known that stimulating the SC can elicit eye movements.

Onset times of visually guided saccades have a bimodal distribution. The faster type of saccades are termed `express saccades'. Ablation of the SC but not of the FEF makes express saccades disappear.

Eye movements are important for visual consciousness.

FEF and LIP stimulation elicits saccadic eye and head movements.

The contribution of head-saccades to full saccades can be influenced by knowledge about the target of the next saccade.

Brainstem activation is very similar to actual muscle behavior.

According to King, the principal function of the SC is initiating gaze shifts.

Before a saccade is made, the region that will be the target of that saccade is perceived with higher contrast and visual contrast.

In the first experiment by Brouwer et al, people fixated different parts of a shape depending on whether the task was just to look at it or grasp it.

The subject's initial saccade, however, was not influenced by the task.

The SC is also involved in eye, head, whole-body, ear, whisker and other body movements.

Receptive fields in some LIP neurons shift just before a saccade to where their usual receptive field will be after the saccade.

Correct pro-saccades are executed earlier than correct anti-saccades.

In saccade/anti-saccade experiments, direction errors are confined to the anti-saccade condition.

In anti-saccade experiments, incorrect saccades (those in the direction of the visual stimulus) occur earlier after target onset than do correct anti-saccades.

The timing of correct pro-saccades has a bi-modal distribution. One class of pro-saccades happens very fast (express saccades), the others take a little longer.

Express saccades are thought of as reflex behavior. The reflex behind them is referred to as the 'visual grasp reflex'.

They are believed to be the result of a direct translation of a visual stimulus into a motor command.

In anti-saccade conditions, the `visual grasp reflex' must be suppressed.

One family of models for saccades and anti-saccades are the `accumulator models'.

These models pose that activation of saccade and saccade suppression neurons race each other. The one first to reach a threshold wins.

Activation of FEF and SC neurons is higher before direction error saccades in anti-saccade tasks than before correct anti-saccades.

Munoz and Everling assume that there are distinct populations of fixation and saccade neurons in the SC and FEF.

In a more recent paper, Casteau and Vitu state that there is some debate about that. However, they, too argue for distinct fixation neurons. On the other hand, they also state that fixation neurons probably are not located in the SC itself, which is in contrast of what Munoz and Everling write.

Omnipause neurons in the reticular formation tonically inhibit `the saccade-generation circuit'.

It seems unclear what is the original source of SC inhibition in preparation of anti-saccades. Munoz and Everling cite the supplementary eye fields (SEF), dorsolateral prefrontal cortex (DLPFC) as possible sources, and the substantia nigra pars reticulata (SNpr).

Saccade targets tend to be the centers of objects.

When reading, preferred viewing locations (PVL)—the centers of the distributions of fixation targets---are typically located slightly left of the center of words.

When reading, the standard deviation of the distribution of fixation targets within a word increases with the distance between the start and end of a saccade.

Saccades are thought to be biased toward a medium saccade length; long saccades typically undershoot, short saccades overshoot.

During reading, the further a saccade lands from the center of a word, the greater the probability of a re-fixation.

Pajak and Nuthmann found that saccade targets are typically at the center of objects. This effect is strongest for large objects.

It is the reticular formation that initiates saccades.